That’s a huge move for a currency, even for the volatile pound, which then recovered most of that ground to trade at $1.2365. Market watchers were baffled by the move, which they blamed on various factors including a trader’s “fat finger” mistake, a rogue automated trading algorithm making decisions based on news websites or social media; or comments made by France’s president, Francois Hollande. Hollande said “Britain wants to leave but not pay anything. interview skills by qasim ali shahThat’s not possible.” His comments suggest he will push for a hard stance in negotiating Britain’s exit, known as Brexit, from the bloc’s tariff-free single market. The comments were made hours before the flash crash, but may have had an effect on trading only when Asian markets opened. Some speculated the crash could have been caused by a combination of these reasons. Kathleen Brooks, research director at Forex.com and City Index, said that computerized trading programs that scan news sites could have been prompted to start selling pounds by the increase in negative headlines about Britain. Once the pound started moving lower, she said, other computerized trading programs could have followed suit, compounding the drop. The crash occurred during a “twilight period” between major market trading hours – after the close of markets in the U.S. and just as Asian traders were starting their day.
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Plaxgen is developing the StatRes Test, which predicts patient statin response based on a serum test performed in advance of the first prescription. While the enzymatic effect of statins on inhibiting cholesterol synthesis in the liver is already well-characterized, it is not fully understood how statins change blood cholesterol composition from low-density lipoproteins to high density. In addition, patient responses differ to differentstatins, and physicians must currently apply a trial and error approach to find the right match. Besides, 5-20% of patients do not respond to statins at all. This important research shows, for the first time, a mechanism-of-action whereby statins interact directlywiththe cholesterol particle and change its distribution from low-density to high-density particles, said Shanmugavel Madasamy,PhD, lead studyauthorand Plaxgen CEO. We believe this non-enzymatic mechanism could explain how statins modulate the formation of HDL particles in the plasma, which has never been well-understood, and help in the further development of our StatRes test, that matches patients to statins via a blood test. We have shown, for the first time, that there are non-enzymatic mechanisms involved that could have an impact on predicting statin efficacy, said Alan H.B. Wu, PhD, Professor Laboratory Medicine, University of California, San Francisco; Chief, Clinical Chemistry Laboratory, San Francisco General Hospital, and a co-author of the study. Until now, the prevailing view of the mechanism of action for statins has been associated to inhibition of HMG-CoA reductase, a key step in the formation of cholesterol. The flow cytometry and serum-based screening assay, if developed further, has a very strong potential to become an assay of choice for screening patients for statintherapy, said Dr. J. Paul Robinson,PhD, Professor of Biomedical Engineering and Director, CytometryLaboratories, Purdue University. To view the paper, please go here: http://www.ajconline.org/article/S0002-9149(16)31244-9/fulltext?rss=yes About Plaxgen Privately-held Plaxgen is developing diagnostics in atherosclerosis, disease, and other plaque-related diseases, using its proprietary Plaque Array technology.http://youtu.be/Pf7JuJcVzQU
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